Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
9377701 | Biological Psychiatry | 2005 | 4 Pages |
Abstract
Our data indicate a potentially pathophysiologic decrease of serum Aβ1-42 antibodies in AD. Amyloid β1-42 antibodies in the serum alone, however, seem not to be useful as a diagnostic marker of AD.
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Authors
Sylvia Brettschneider, Nils G. Morgenthaler, Stefan J. Teipel, Christina Fischer-Schulz, Katharina Bürger, Richard Dodel, Yansheng Du, Hans-Jürgen Möller, Andreas Bergmann, Harald Hampel,