| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9423089 | Brain Research Reviews | 2005 | 12 Pages |
Abstract
The latest published reports on NO and neurogenesis indicate that NO physiologically participates in the control of adult neurogenesis by modulating the proliferation and fate of the SVZ progenitor cells. These effects might be partially due to a direct inhibition of the EGFR by S-nitrosylation.
Keywords
DETA-NONeuronal specific enolaseEGFS-nitroso-N-acetylpenicillamineNSEGFAPEPOSVZNOSEGFRPSA-NCAMl-NAMENω-nitro-l-arginine methyl estererythropoietinBrdUbromodeoxyuridineDevelopment and regenerationCell proliferationdentate gyrusSNAPepidermal growth factorsubventricular zoneadult neurogenesisNitric oxidenitric oxide synthaseGlial fibrillary acidic proteinGenesis of neurons and gliaNeural precursorEpidermal growth factor receptor
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Authors
Esperanza R. Matarredona, Maribel Murillo-Carretero, Bernardo Moreno-López, Carmen Estrada,