Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
9425861 | Neuroscience | 2005 | 7 Pages |
Abstract
In summary, in animals exposed to nicotine throughout the prenatal period, but not in unexposed animals, respiratory bursts that occur during hypoxia dramatically increase the frequency of excitatory synaptic events in cardiac vagal neurons. This study establishes a likely neurochemical mechanism for the heart rate responses to hypoxia and a link between prenatal nicotine exposure and exaggerated bradycardia responses during hypoxia that may contribute to sudden infant death syndrome.
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Neuroscience (General)
Authors
C. Evans, J. Wang, R. Neff, D. Mendelowitz,