| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9426223 | Neuroscience | 2005 | 14 Pages |
Abstract
To investigate the mechanisms involved in the change of expression of these proteins following axotomy, the facial nerve was perfused with tetrodotoxin for 8 days. The blockade of action potential significantly decreased GLuR2-3, NR1and NR2A mRNAs in the ipsilateral facial nuclei. Thus, axotomy-induced changes in mRNA abundance seemed to depend partly on disruption of activity.
Keywords
PBSPSD95TBSRODRAFAMPARPFAvesicular inhibitory amino acid transporterTTXMVNVGLUT2Cy3NMDARTBSTexcitatory amino acidsstandard deviationbeta cateninTris buffer salinetetrodotoxinrelative optical densityPost-synaptic densityvesicular glutamate transporter 2Cyanine 3Phosphate-buffered salinemedial vestibular nucleusVIAATparaformaldehydecadherinN-methyl-d-aspartate receptor
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Authors
L. Eleore, I. Vassias, P.-P. Vidal, C. de Waele,