| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9436492 | Hearing Research | 2005 | 8 Pages |
Abstract
Isolated OHCs respond with slow shortening and consutive cell stiffness increase to mechanical insult. This phenomenon seems operating with calcium-, and phosphorylation-dependent modifications of the cytoskeletal proteins. The subsequent electromotility gain decrease suggests a slow OHC shortening driven regulation of the cochlear amplifier with simultaneous safety control of the auditory periphery against overstimulation.
Keywords
Related Topics
Life Sciences
Neuroscience
Sensory Systems
Authors
Rezsö Borkó, Tamás József Batta, István Sziklai,