Article ID Journal Published Year Pages File Type
9872122 International Journal of Radiation Oncology*Biology*Physics 2005 9 Pages PDF
Abstract
Conclusions: Celecoxib strongly enhanced the sensitivity of HN5 carcinoma cells to radiation, which, mechanistically, can be attributed to the inhibition of DNA repair processes in radiation-damaged cells.
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Related Topics
Physical Sciences and Engineering Physics and Astronomy Radiation
Preview
Inhibition of DNA repair as a mechanism of enhanced radioresponse of head and neck carcinoma cells by a selective cyclooxygenase-2 inhibitor, celecoxib
Authors
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