| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9913288 | Immunobiology | 2005 | 6 Pages |
Abstract
Thus, aCL/β2GPI plays dual roles in the pathogenesis of APS, firstly by deranging the fibrinolytic system and secondly by activating monocytes, endothelial cells and thrombocytes to produce TF or thromboxane.
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Authors
Shinsuke Yasuda, Miyuki Bohgaki, Tatsuya Atsumi, Takao Koike,