| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9921400 | European Journal of Pharmacology | 2005 | 7 Pages |
Abstract
The pharmacological and biochemical data presented in this study can be explained quantitatively by a cell compartment model in which PDE 3 appears to be colocalized with the contractile machinery responsible for the effects of glucagon but not those of dobutamine. Neither PDE 1 nor PDE 2 appears to regulate the inotropic effects of dobutamine and glucagon in rat ventricular myocardium.
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Authors
MarÃa Jesús Juan-Fita, MarÃa Luisa Vargas, Jesús Hernández,