| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9942475 | The American Journal of the Medical Sciences | 2005 | 7 Pages |
Abstract
Hypercalciuria and hypermagnesuria accompany aldosteronism and account for a decline in their plasma ionized concentrations and secondary hyperparathyroidism with bone resorption. Attenuation of bone loss in aldosteronism can be achieved with hydrochlorothiazide; however, mono- and divalent cation homeostasis, together with bone integrity, are each preserved with the combination hydrochlorothiazide + spironolactone.
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Authors
Aliye L. Runyan, Vikram S. MD, Yao MD, PhD, Karl T. MD, John W. MD, Syamal K. PhD,