Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
10215273 | Trends in Cardiovascular Medicine | 2018 | 43 Pages |
Abstract
Collectively, the completion of the Human Genome Project and subsequent development of high-throughput next-generation sequencing methodologies have revolutionized genomic research. However, the rapid sequencing and analysis of thousands upon thousands of human exomes and genomes has taught us that most genes, including those known to cause heritable cardiovascular disorders such as long QT syndrome, harbor an unexpected background rate of rare, and presumably innocuous, non-synonymous genetic variation. In this Review, we aim to reappraise the genetic architecture underlying both the acquired and congenital forms of long QT syndrome by examining how the clinical phenotype associated with and background genetic variation in long QT syndrome-susceptibility genes impacts the clinical validity of existing gene-disease associations and the variant classification and reporting strategies that serve as the foundation for diagnostic long QT syndrome genetic testing.
Keywords
IKAChcLQTSTorsades de pointesEADABSQTcAMPTDPACMGLQTSSCDMAFCa2+DNANa+VUSArrhythmiaGenetic testingdeoxyribonucleic acidECGelectrocardiogramearly afterdepolarizationGenetic variationSodiumLong QT syndromeminor allele frequencySudden cardiac deathGenome-wide association studyGWASVariant of uncertain significancePotassiumAmerican College of Medical Genetics and GenomicsCalcium
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Authors
John R. Giudicessi, Arthur A.M. Wilde, Michael J. Ackerman,