Article ID Journal Published Year Pages File Type
10307727 Schizophrenia Research 2014 6 Pages PDF
Abstract
These data suggest that increased GRK5 expression may contribute to the pathophysiology of schizophrenia via abnormal regulation of the cytoskeleton, endocytosis, signaling, GPCRs, and histone modification.
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Related Topics
Life Sciences Neuroscience Behavioral Neuroscience
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Increased G protein-coupled receptor kinase (GRK) expression in the anterior cingulate cortex in schizophrenia
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