Article ID Journal Published Year Pages File Type
10754652 Biochemical and Biophysical Research Communications 2014 7 Pages PDF
Abstract
The interaction between M. catarrhalis and EGFR increases airway inflammation caused by this pathogen. Our data suggest that the inhibition of EGFR signaling in COPD could be an interesting target for reducing M. catarrhalis-induced airway inflammation.
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Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Preview
The Moraxella catarrhalis-induced pro-inflammatory immune response is enhanced by the activation of the epidermal growth factor receptor in human pulmonary epithelial cells
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