Article ID Journal Published Year Pages File Type
10755329 Biochemical and Biophysical Research Communications 2014 6 Pages PDF
Abstract
Hence, UPR is activated following skin injury, and functionally connected to the production of proinflammatory mediators. In addition, prolongation of UPR in diabetic non-healing wounds aggravates ER stress and weakens the angiogenic phenotype of wound macrophages.
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Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Preview
Deregulated unfolded protein response in chronic wounds of diabetic ob/ob mice: A potential connection to inflammatory and angiogenic disorders in diabetes-impaired wound healing
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