Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
10757072 | Biochemical and Biophysical Research Communications | 2014 | 7 Pages |
Abstract
Neovessel formation (angiogenesis) accelerates liver fibrosis at least in part via provision of latent TGF-β that activated on the surface of HSCs by PLK, thereby resultant active TGF-β stimulates the activation of HSCs.
Keywords
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Authors
Kotaro Sakata, Satoshi Eda, Eun-Seo Lee, Mitsuko Hara, Masaya Imoto, Soichi Kojima,