| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10757327 | Biochemical and Biophysical Research Communications | 2013 | 6 Pages |
Abstract
- SAR induced apoptosis via the caspase-dependent mitochondrial apoptosis pathway.
- SAR activated UPR signaling pathways.
- SAR provoked the generation of ROS.
- The generation of ROS was necessary for ER stress mitochondrial apoptotic pathways.
- Sarsasapogenin (SAR) retarded the cell cycle at S and G2 phase.
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Authors
Shuying Shen, Yi Zhang, Rui Zhang, Xingguo Gong,