Article ID Journal Published Year Pages File Type
10761560 Biochemical and Biophysical Research Communications 2012 6 Pages PDF
Abstract
► NO induced MAPK-ERK1/2 activation during myocyte reoxygenation injury. ► ERK activation is required for the increased and stabilization of XIAP. ► XIAP inhibits reoxygenation induced apoptosis in post-mitochondrial manner.
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Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Preview
Nitric oxide preserves XIAP and reduces hypoxia/reoxygenation-induced cardiomyocytes apoptosis via ERK1/2 activation
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