| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10762503 | Biochemical and Biophysical Research Communications | 2011 | 5 Pages |
Abstract
⺠In this study we present Mnl2 as a new component of the ERAD pathway. ⺠We propose that Mnl2 cooperates with Mnl1 in the generation of the glycan degradation signal. ⺠The recognition particle Yos9 cannot recognize the glycan structure of the ERAD substrate CPY* in the absence of Mnl1 and Mnl2. ⺠Mnl2 does not affect the degradation of unglycosylated substrate CPY*0000.
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Authors
Elena Martinez Benitez, Alexandra Stolz, Alexander Becher, Dieter H. Wolf,