| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10764815 | Biochemical and Biophysical Research Communications | 2010 | 6 Pages |
Abstract
⺠Neuronal nitric oxide synthase (nNOS) increases oxidative stress after lung injury and sepsis. ⺠nNOS-gene deficiency prevents the development of oxidative/nitrosative stress in the lung. ⺠However, nNOS-gene deficiency does not improve survival in this murine sepsis model.
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Authors
Matthias Lange, Yoshimitsu Nakano, Daniel L. Traber, Atsumori Hamahata, Aimalohi Esechie, Collette Jonkam, Kamna Bansal, Lillian D. Traber, Perenlei Enkhbaatar,