| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10799732 | Biochimica et Biophysica Acta (BBA) - General Subjects | 2016 | 31 Pages |
Abstract
These findings confirm previously published data obtained in cell cultures and strengthen the hypothesis of different mechanism of action of the two MFN2 mutations. Considering the number of neurodegenerative diseases associated to mitochondrial dynamics, the Tg(hb9:MTS-Kaede) zebrafish line is a promising model to study in vivo alterations of mitochondrial transport underlying human diseases.
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Authors
Giorgia Bergamin, Domenico Cieri, Giovanni Vazza, Francesco Argenton, Maria Luisa Mostacciuolo,