| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10800302 | Biochimica et Biophysica Acta (BBA) - General Subjects | 2014 | 9 Pages |
Abstract
These data support that AMPK activation may be an important mechanism for thiazolidinediones preserving β-cell function.
Keywords
ACCGSISTZDKATPIFGPPARγshRNAIGTAMPKAMP-activated protein kinaseshort hairpin RNAimpaired glucose toleranceImpaired fasting glucoseacetyl-CoA carboxylaseGlucose-stimulated insulin secretionTroglitazonethiazolidinedionesBeta-cellATP-sensitive K+ channelVoltage-dependent potassium channelperoxisome proliferator-activated receptor γ
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Authors
Ruyuan Deng, Aifang Nie, Fangfang Jian, Yun Liu, Hongju Tang, Juan Zhang, Yuqing Zhang, Li Shao, Fengying Li, Libin Zhou, Xiao Wang, Guang Ning,