| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10800558 | Biochimica et Biophysica Acta (BBA) - General Subjects | 2013 | 8 Pages |
Abstract
⺠Depletion of calcium by transfer of cells to S-MEM destroyed adherens junctions. ⺠Nuclear translocation of the Ah receptor induced CYP1A1 and Slug mRNAs. ⺠β-Catenin was released from E-cadherin complexes by destruction of adherens junctions. ⺠Interaction between β-catenin and the Ah receptor increased after S-MEM treatment. ⺠Knockdown of β-catenin increased the level of Ah receptor mRNA.
Keywords
GAPDHMAFCytochrome P-450 1A1HSP90XREFOXM1TCDDCYP1A1DAPTbHLHARNT2,3,7,8-Tetrachlorodibenzo-p-dioxinE-cadherinN-[N-(3,5-difluorophenacetyl)-L-alanyl]-S-phenylglycine t-butyl esterPer-Arnt-Simbasic helix-loop-helixβ-cateninEMTforkhead box M1lymphoid enhancer factorxenobiotic-responsive elementAh receptor nuclear translocatorLefPASglyceraldehyde-3-phosphate dehydrogenasearyl hydrocarbon receptor
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Authors
Shuya Kasai, Takanori Ishigaki, Ryo Takumi, Toru Kamimura, Hideaki Kikuchi,