| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10804217 | Biochimie | 2011 | 7 Pages |
Abstract
⺠Kininogen (HK) binding to endothelial cells (RAEC) was abolished by heparin/Zn+2. ⺠Surprisingly, prekallikrein (PK) interaction with RAEC was not modified by heparin. ⺠BK release, from HK hydrolysis, increased in the presence of heparin/Zn+2.
Keywords
ECMHuPKFXIIHEPPrekallikreinRAECECV304BDSC4SC6STDSbradykininBiotinEndothelial cellsKeratan sulfateFactor XIIExtracellular matrixHeparan sulfateHeparinLow molecular weight heparinhuman plasma kallikreinchondroitin 4-sulfateChondroitin 6-sulfateKininogenhigh molecular weight kininogenGlycosaminoglycansZinc ions
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Authors
Andrezza J. Gozzo, Guacyara Motta, Ilana Cruz-Silva, Viviane A. Nunes, Nilana M.T. Barros, Adriana K. Carmona, Misako U. Sampaio, Yara M.C. Michelacci, Kazuaki Shimamoto, Helena B. Nader, Mariana S. Araújo,