| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10816229 | Cellular Signalling | 2013 | 9 Pages |
Abstract
⺠Syncytin-1 knockdown significantly inhibited BeWo cell growth and DNA synthesis. ⺠Syncytin-1 overexpression promoted CHO cell proliferation. ⺠Syncytin-1 could promote the G1/S transition through p15 and CDK4. ⺠Syncytin-1 may co-regulate the cytotrophoblast “pool” through its dual functions.
Keywords
IUGRPCNASyncytin-1SyncytiumASCTSyncytiotrophoblastTGFHCsDEPCGAPDHCDK3-(4, 5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide5-bromo-2′-deoxyuridineMTTProliferating Cell Nuclear AntigenG1/S transitionHCGChoBrdUtransforming growth factorChinese Hamster OvaryTrophoblastPlacentadiethylpyrocarbonateFusionintrauterine growth restrictionPreeclampsiaCell cyclecyclin dependent kinasehuman chorionic gonadotropin
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Authors
Qiang Huang, Jinping Li, Fengchao Wang, Matthew T. Oliver, Tracy Tipton, Ya Gao, Shi-Wen Jiang,