| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10838199 | Pharmacology Biochemistry and Behavior | 2011 | 7 Pages |
Abstract
⺠Endocannabinoid system modulates DOI-induced head-twitch response (HTR).⺠AMPA receptor antagonist inhibits DOI-induced HTR.⺠AMPA receptor positive modulator reverses inhibition of HTR by NBQX and URB597.⺠Endocannabinoid and glutamatergic systems participate in the mechanism of DOI.
Keywords
DOIα-amino-3-hydroxy-5-methyl-4-isoxazolepropionate2,5-dimethoxy-4-iodoamphetamineFAAHMK-801CB2N-methyl-d-aspartateNMDACB1NBQX1,2,3,4-tetrahydro-6-nitro-2,3-dioxo-benzo[f]quinoxaline-7-sulfonamideAM404AMPAURB597PCPHTRDOBmGluLSDTHCTRPV1N-(piperidin-1-yl)-5-(4-iodophenyl)-1-(2,4-dichlorophenyl)-4-methyl-1H-pyrazole-3-carboxamide5-HTAM281arachidonylethanolamide∆9-tetrahydrocannabinolanandamideAttention-Deficit/Hyperactivity DisorderFatty acid amide hydrolaseLysergic acid diethylamideBINAADHDTheanineanalysis of varianceANOVAdizocilpineBehaviorSerotoninPhencyclidineCSFCerebrospinal fluidcannabinoid receptor type 1head-twitch responseTransient receptor potential vanilloid 1CannabinoidsglutamateMetabotropic glutamateAMPA receptorSerotonin receptorcannabinoid receptor type 2
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Authors
Nobuaki Egashira, Atsunori Shirakawa, Ryoko Okuno, Kenichi Mishima, Katsunori Iwasaki, Ryozo Oishi, Michihiro Fujiwara,