| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 1947937 | Biochimica et Biophysica Acta (BBA) - General Subjects | 2010 | 13 Pages |
Abstract
In this study, we have identified NO depletion to be an important factor in promoting premature hemolysis with the progress of leishmanial infection. The study implicates NO to be a possible target for future drug development towards the promotion of erythrocyte survival in VL.
Keywords
FITCNBD-PCVisceral leishmaniasis (VL)VCl3SAGPrx2ISDTrxDAF-2DAS-nitrosoglutathioneGSNOFCShpfDMFN-ethylmaleimideNADPHEMA4,5-diaminofluorescein diacetateErythrophagocytosisIsosorbide dinitratethioredoxindimethylformamidesodium stibogluconatefoetal calf serumPhosphatidylserinefluorescein isothiocyanateVisceral leishmaniasisNeddNEMNitric oxideNitric oxide (NO)nicotinamide adenine dinucleotide phosphate reduced formCalcium homeostasiscaspase 3
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Authors
Kaustav Dutta Chowdhury, Gargi Sen, Tuli Biswas,