| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 1968577 | Clinical Biochemistry | 2016 | 5 Pages |
Abstract
Abnormal redox metabolism takes place in DS individuals. Reducing GPx3 may be a compensatory mechanism of protection against intracellular OS. Moreover, monitoring of decreases in GPx3 activity may be a useful biomarker for evaluating OS in DS patients.
Keywords
eNOSMDAGPxsLyMnNOSHCsRNSiNOSNOSCATneuronal NOSinducible NOSROSPolyunsaturated fatty acidsPUFAstandard deviationendothelial NOSOxidative stressneuDown syndromesuperoxide dismutasesLymphocytemalondialdehydeBiomarkersNeutrophilsNitric oxidenitric oxide synthaseSODsC reactive proteinCRPCatalaseGlutathione peroxidasesreactive nitrogen speciesReactive oxygen species
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Authors
Jingyi He, Tingyu Li, Jie Chen, Youxue Liu, Feng Xiong, Jing Yang, Cui Song,