| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 1988895 | Journal of Chemical Neuroanatomy | 2013 | 8 Pages |
Abstract
Thus, postnatal AE causes motoneuronal loss in the XIIn-v. This may compromise upper airway control and contribute to increased risk of upper airway obstructions and sudden infant death in FASD victims.
Keywords
α-Adrenergic receptorsSIDSFASDDMSOFetal alcohol spectrum disordersFetal alcohol spectrum disorders (FASD)Dimethyl sulfoxidegestational daypostnatal daySudden Infant Death SyndromeSudden Infant Death Syndrome (SIDS)alcohol exposurehypoglossal nucleusChATcholine acetyltransferaseAlpha-1 adrenergic receptorserotonin 2A receptorSerotonin receptors
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Authors
Georg M. Stettner, Leszek Kubin, Denys V. Volgin,