Article ID Journal Published Year Pages File Type
2862559 American Journal of Hypertension 2007 8 Pages PDF
Abstract
The increased store-operated and second messenger-operated calcium influx through TRPC3 channels in monocytes from SHRs may be responsible for a more aggressive effect in promoting vascular disease in genetic hypertension.
Related Topics
Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Authors
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