Article ID Journal Published Year Pages File Type
5506006 Biochemical and Biophysical Research Communications 2017 7 Pages PDF
Abstract
In conclusion, SCFAs lowered TNF-α−induced MCP-1 expression by reducing phosphorylation of p38 and JNK in a GPR41/43-dependent manner in HRCEs, suggesting that SCFA modification may be a new therapeutic tool for preventing progression of renal inflammation and fibrosis.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Authors
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