| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5506922 | Biochemical and Biophysical Research Communications | 2016 | 20 Pages |
Abstract
High expression of HOTAIR promoted the onset of ischemic infarct induced by hypoxia. Moreover, the finding showed that HOTAIR promoted ischemic infarct induced by hypoxia through regulating NOX2 expression, which could add our understanding of the molecular mechanisms in ischemic infarct.
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Authors
Lei Yang, Zu-Neng Lu,