| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5513786 | Microvascular Research | 2017 | 6 Pages |
Abstract
Artificially applied c-kit+ cells interact with the target organ endothelium following IR injury. This interaction seems to depend on TLR-MyD88 signaling. Therapeutic blockade of TLR signaling for anti-inflammatory purposes might interfere with regenerative cell-based therapy protocols.
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Authors
Peter Donndorf, Saifullah Abubaker, Brigitte Vollmar, Christian Rimmbach, Gustav Steinhoff, Alexander Kaminski,