| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5823510 | Biochemical Pharmacology | 2013 | 10 Pages |
Abstract
Renal mitochondria from the remnant kidney of uninephrectomized rats undergo hypertrophy, characterized by increased respiration, enhanced formation of reactive oxygen species, but compensatory changes in glutathione status.
Keywords
tBHKHBJC-1OGCDAPIantimycin ART-PCRGAPDHGSHNPXNRKDICMVK2-Oxoglutarate carrier4,6-diamidino-2-phenylindole dihydrochlorideBSAbovine serum albuminKrebs-Henseleit bufferOxidative stressdicarboxylate carrierUninephrectomylactate dehydrogenaseLDHproximal tubularMethyl vinyl ketoneMitochondriatert-butyl hydroperoxidereverse transcription-polymerase chain reactionnormal rat kidneyGlutathioneglyceraldehyde 3-phosphate dehydrogenase
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Authors
Bavneet Benipal, Lawrence H. Lash,