| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5997009 | Progress in Cardiovascular Diseases | 2011 | 9 Pages |
Abstract
In conclusions, the studies differed substantially in both the cardiovascular outcomes examined and the polymorphisms examined, so there is little confirmation of results across cohorts. Gene-environment interaction studies can help explore the mechanisms and the potential pathway in the association between air pollution and a cardiovascular outcome; replication of findings and studies involving multiple cohorts would be needed to draw stronger conclusions.
Keywords
LPLGSSGSTM1TLR4Ptgs2DGCR8NASLVMACEMTHFRHFEALOX15NQO1HMOX-1ADRB2GSTP1GSTT1AGTR1type 1 angiotensin II receptorarachidonate 15-lipoxygenaseIL-6EDN1ITPR2NAD(P)H dehydrogenase, quinone 1PTGS1HRVROSMyocardial infarctionAngiotensin-I Converting EnzymeAngiotensinogenApoeapolipoprotein Eendothelin 1interleukin 6cardiovascular diseasegene-environment interactionLeft ventricular massparticulate matterCVDVascular endothelial growth factorVascular Endothelial Growth Factor (VEGF)Blood pressureLipoprotein lipaseMESAMethylenetetrahydrofolate reductaseGenome-wide association studyGWASMicroRNAMiRNAheart rate variabilityAGTheme oxygenase 1HemocromatosisC-reactive proteinCRPprostaglandin-endoperoxide synthase 2PolymorphismSingle-nucleotide polymorphismSNPBlack carbonReactive oxygen speciesβ2-adrenergic receptorToll-like receptor 4
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Antonella Zanobetti, Andrea Baccarelli, Joel Schwartz,