| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6119872 | Journal of Clinical Virology | 2016 | 7 Pages |
Abstract
Taken together, our results suggest that V3 DII evolution may differ in strains associated with HCC occurrence. The presence of an intrinsically “disordered” V3 duplicate may alter the NS5A protein network. Further investigations are necessary to elucidate the potential impact of V3 duplication in the context of carcinogenesis.
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Authors
Odile Petsaris, Sophie Vallet, Hélène Le Guillou-Guillemette, Pascal Veillon, Stéphanie Gouriou, Georges Barbier, Jean-Baptiste Nousbaum, Philippe Saliou, Gisèle NKontchou, Jean-Claude Trinchet, Francoise Lunel-Fabiani, Christopher Payan,