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Lipidomics reveals dysfunctional glycosynapses in schizophrenia and the G72/G30 transgenic mouse

Article ID Journal Published Year Pages File Type
6824683 Schizophrenia Research 2014 5 Pages PDF
Abstract
Our data suggest that structural lipid alterations in oligodendrocyte glycosynapses are responsible for dysconnectivity in schizophrenia and that increased expression of G72 protein may play a role in the development of abnormal glycosynapses.
Keywords
SchizophreniaSulfatidesDysconnectivityPlasmalogensGlycosynapse
Related Topics
Life Sciences Neuroscience Behavioral Neuroscience
Preview
Lipidomics reveals dysfunctional glycosynapses in schizophrenia and the G72/G30 transgenic mouse
Authors
Paul L. Wood, Michaela D. Filiou, David M. Otte, Andreas Zimmer, Christoph W. Turck,
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Journal
Schizophrenia Research
Journal: Schizophrenia Research
Related Categories
Schizophrenia
Sulfatides
Dysconnectivity
Plasmalogens
Glycosynapse
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