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Glial cell-derived neurotrophic factor alleviates sepsis-induced neuromuscular dysfunction by decreasing the expression of γ- and α7-nicotinic acetylcholine receptors in an experimental rat model of neuromyopathy

Article ID Journal Published Year Pages File Type
8294640 Biochemical and Biophysical Research Communications 2018 22 Pages PDF
Abstract
GDNF administration may improve patient outcomes by reducing the demyelination of nerves and the expression of γ- and α7-nAChR.
Keywords
Neuromuscular dysfunctionSepsisnicotinic acetylcholine receptor
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Preview
Glial cell-derived neurotrophic factor alleviates sepsis-induced neuromuscular dysfunction by decreasing the expression of γ- and α7-nicotinic acetylcholine receptors in an experimental rat model of neuromyopathy
Authors
Xin Wang, Su Min, Fei Xie, Jun Yang, Liang Li, Jingyuan Chen,
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Journal
Biochemical and Biophysical Research Communications
Journal: Biochemical and Biophysical Research Communications
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