Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
8297816 | Biochemical and Biophysical Research Communications | 2013 | 5 Pages |
Abstract
Exposure to Ang II, a mediator of anoxic injury, initiates a rapid autophagy response, perhaps in an attempt to protect tissues from the impending noxious effects. However, when anoxia (and thereby release of Ang II) is prolonged, the process of apoptosis dominates. These processes will determine the outcome of cardiomyocyte well-being in states of hypoxia.
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Authors
Xianwei Wang, Yao Dai, Zufeng Ding, Magomed Khaidakov, Federico Mercanti, Jawahar L. Mehta,