Article ID Journal Published Year Pages File Type
8304165 Biochimie 2018 28 Pages PDF
Abstract
Upregulation of Egr2 and Egr4 could demote the activation of JNK/c-JUN pathway and the expression of pro-inflammatory cytokines in MCAO rats, so that Egr2 and Egr4 might be potential targets for ischemic stroke in future.
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Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry
Preview
Overexpression of Egr2 and Egr4 protects rat brains against ischemic stroke by downregulating JNK signaling pathway
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