| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8313570 | Clinica Chimica Acta | 2013 | 7 Pages |
Abstract
⺠Four mechanisms are predicted to enhance functions or targeted therapy of HDL. ⺠Caveolae recruit the HDL signal to bind their receptors. ⺠SR-BI mediates anchoring and fluidity for the signal-lipid of HDL. ⺠LCAT concentrates the signaling lipids at the surface of the HDL particle. ⺠MicroRNAs are delivered in blood to special targets by HDL.
Keywords
FingolimodmiRNAsnSMase2HDLPGI2FTY720LCATS1PRsCOX-2S1PApoA-ISR-BIeNOSVLDLhigh-density lipoproteinSmall interfering RNAsiRNAApolipoprotein A-Isphingosine 1-phosphatesphingomyelinaseBehçet's diseaseCaveolaeTargeted therapymicroRNAsBlood–brain barrierBBBendothelial nitric oxide synthaseCyclooxygenase-2lecithin-cholesterol acyltransferasevery low-density lipoproteinLow-density lipoproteinLDLProstaglandin I2free cholesterolSphingosine 1-phosphate receptorsscavenger receptor class B type I
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Authors
Xing Liu, Rong Suo, Sheng-Lin Xiong, Qing-Hai Zhang, Guang-Hui Yi,