Neuroinflammation and endoplasmic reticulum stress are coregulated by cyclo(His-Pro) to prevent LPS neurotoxicity

Article ID	Journal	Published Year	Pages	File Type
8323587	The International Journal of Biochemistry & Cell Biology	2014	11 Pages	PDF

Keywords

TNF HO-1 GAPDH Nrf2 COX-2 iNOS DCFH-DA UPR GFAP eIF2α IBA1 CHP TLR4 IL-1 tunicamycin LPS 2′,7′-Dichlorofluorescein-diacetate TNFα Gp91phox NF-κB BiP C/EBP-homologous protein CCm ROS Ara C cyclooxygenase amyotrophic lateral sclerosis AIDS interleukin 1 beta Alzheimer's disease ALS Parkinson's disease Fourier transform infrared tumor necrosis factor alpha CHOP CNS RONS inducible nitric oxide synthase acquired immune deficiency syndrome central nervous system endoplasmic reticulum FTIR eukaryotic translation initiation factor 2 lipopolysaccharide Multiple sclerosis ionized calcium binding adapter molecule 1 Nitric oxide heme oxygenase 1 Unfolded protein response glial fibrillar acidic protein glyceraldehyde-3-phosphate-dehydrogenase Reactive oxygen/nitrogen species Reactive oxygen species Toll-like receptor 4

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Biochemistry

Neuroinflammation and endoplasmic reticulum stress are coregulated by cyclo(His-Pro) to prevent LPS neurotoxicity

Authors

Ilaria Bellezza, Silvia Grottelli, Anna Lisa Mierla, Ivana Cacciatore, Erika Fornasari, Luca Roscini, Gianluigi Cardinali, Alba Minelli,