| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8339900 | Journal of Traditional Chinese Medical Sciences | 2017 | 10 Pages |
Abstract
The possible mechanism of YQHX prescription for preventing and treating post-MI myocardial remodeling may function through strengthening the activation AMPK signal pathway by LKB1, thus further increasing the expression of downstream transcription factor proteins and initiating mitochondrial replication and transcription; as a result, the YQHX prescription can improve the post-MI damage in mitochondrial morphological structure in the tissue at heart marginal zone as well as enhance mitochondrial biosynthesis and ATP synthesis.
Keywords
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Authors
Jiangong Wu, Shuwen Guo, Xi Chen, Wenchen Liu, Mingjing Zhao, Lu Zhang, Zhaoduo Su, Yuqin Zhang,