| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8361600 | Regulatory Peptides | 2011 | 8 Pages |
Abstract
⺠Mechanisms for sympathetic overactivity in chronic kidney disease are unclear. ⺠High parathyroid hormone (PTH) levels are associated with chronic kidney disease. ⺠PTH facilitates norepinephrine release in human atrium and kidney. ⺠Norepinephrine release is PTH1-receptor but not PTH2-receptor dependent. ⺠A splice variant of the PTH2-receptor was found to be expressed in human kidney.
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Authors
S.A. Potthoff, A. Janus, H. Hoch, M. Frahnert, P. Tossios, D. Reber, M. Giessing, H.M. Klein, E. Schwertfeger, I. Quack, L.C. Rump, O. Vonend,