Role of endogenous and exogenous nitric oxide, carbon monoxide and hydrogen sulfide in HCT116 colon cancer cell proliferation

Article ID	Journal	Published Year	Pages	File Type
8524319	Biochemical Pharmacology	2018	54 Pages	PDF

Keywords

CBS DETA-NO S-nitroso-N-acetyl-d,l-penicillamine NG-methyl-l-arginine ZnPPIX 3-bromo-7-nitroindazole 3-MST L-NMMA dl-propargylglycine ODQ PLP AOAA nNOS PAG eNOS int H2S iNOS PI3K CSE 3-(4,5-dimethyl-2-thiazolyl)-2,5-diphenyl-2H-tetrazolium bromide 3-mercaptopyruvate sulfurtransferase L-NIO MTT NAD+aminooxyacetic acid Cell proliferation Apoptosis Bioenergetics Cancer inducible nitric oxide synthase endothelial nitric oxide synthase neuronal nitric oxide synthase Hydrogen sulfide cystathionine-β-synthase cystathionine gamma lyase Signaling Anticancer SNAP phosphoinositide 3-kinase carbon monoxide Nitric oxide nicotinamide adenine dinucleotide heme oxygenase Pyridoxal phosphate Gasotransmitters

Related Topics

Health Sciences Pharmacology, Toxicology and Pharmaceutical Science Pharmacology

Role of endogenous and exogenous nitric oxide, carbon monoxide and hydrogen sulfide in HCT116 colon cancer cell proliferation

Authors

Gabor Oláh, Katalin Módis, Gabor Törö, Mark R. Hellmich, Bartosz Szczesny, Csaba Szabo,