Jageum-Jung improves 2,4-dinitrochlorobenzene-induced atopic dermatitis-like skin lesions in mice and suppresses pro-inflammatory chemokine production by inhibiting TNF-Î±/IFN-Î³-induced STAT-1 and NFÎºB signaling in HaCaT cells

Article ID	Journal	Published Year	Pages	File Type
8532232	Journal of Ethnopharmacology	2018	34 Pages	PDF

Keywords

regulated on activation, normal T-cell expressed and secreted PBS s.e.FBS MDC DNCB DAD ERK 2,4-dinitrochlorobenzene DMSO MAPK Mitogen activated protein kinases thymus and activation-regulated chemokine ELISA Enzyme-linked immunosorbent assay standard error Atopic dermatitis Dinitrochlorobenzene Dimethyl sulfoxide TARC fetal bovine serum Mast cells Phosphate-buffered saline RANTES Nitric oxide JAK Keratinocytes HPLC high-performance liquid chromatography Macrophage-derived chemokine extracellular signal-regulated kinases

Related Topics

Health Sciences Pharmacology, Toxicology and Pharmaceutical Science Pharmacology

Jageum-Jung improves 2,4-dinitrochlorobenzene-induced atopic dermatitis-like skin lesions in mice and suppresses pro-inflammatory chemokine production by inhibiting TNF-Î±/IFN-Î³-induced STAT-1 and NFÎºB signaling in HaCaT cells

Authors

Ju-Hye Yang, Hyun Ju Do, Esther Lee, Nam-Hui Yim, Won-Kyung Cho, Kwang-Il Park, Jin Yeul Ma,