Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
8739752 | Journal of Clinical Virology | 2018 | 6 Pages |
Abstract
The viruses harboring the 245-248 deletion in the neuraminidase gene were still present after discontinuation of oseltamivir treatment and passages in cell cultures, indicating a potential risk for transmission of the deleted virus. Full genome deep sequencing was useful to reveal variant mutations that might be selected due to antiviral treatment, and defective interfering-like viral PB2 RNA in the respiratory samples was detected.
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Authors
Ramona Trebbien, Claus Bohn Christiansen, Thea Kølsen Fischer,