| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9209764 | Medicina Intensiva | 2005 | 7 Pages |
Abstract
The progress described contributes a new importance to oxidative stress and to nitridative stress in the knowledge of the pathogenesis of sepsis, and makes it possible to understand the physiopathological bases of cellular dysfunction in sepsis.
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Authors
F.J. Hurtado Bredda, N. Nin Vaeza, H. Rubbo Amonini,