| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9646402 | Schizophrenia Research | 2005 | 9 Pages |
Abstract
The observed cell membrane deficits in PUFA and essential fatty acid content do not appear to derive from decreased dietary supply. Rather, intrinsic membrane phospholipid metabolism abnormalities may be causative. Overall increased fat intake in schizophrenia patients may contribute to the development of serious medical comorbidities, and further advance the risk for cumbersome metabolic side effects of antipsychotic treatment such as new-onset diabetes mellitus.
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Authors
Martin Strassnig, Jaspreet Singh Brar, Rohan Ganguli,