Article ID Journal Published Year Pages File Type
9940709 American Journal of Hypertension 2005 6 Pages PDF
Abstract
Enalapril and candesartan both specifically inhibit phVEGF-A165 induced myocardial angiogenesis in the normal heart. The mechanism of inhibition is a combination of inhibition of cardiac hVEGF-A expression and of decreased endogenous expression of the mVEGF ligand and receptor system.
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Related Topics
Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Preview
Antagonism of the Renin-Angiotensin System Can Counteract Cardiac Angiogenic Vascular Endothelial Growth Factor Gene Therapy and Myocardial Angiogenesis in the Normal Heart
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