| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9954203 | Nitric Oxide | 2018 | 48 Pages |
Abstract
Metabolites in blood do not reflect NO metabolite status of any organ. Although NO is rapidly inactivated by hemoglobin-mediated oxidation in the circulation, long-lived tissue metabolites may account for the myocardial preconditioning effects of inhaled NO. NO inhalation may afford similar protection in other organs.
Keywords
RSNORNNON-nitrosamineS-nitrosothiolsiNOcGMPAARI/RNOxIschemia-reperfusion injuryReperfusion injuryMyocardial infarctionInhaled nitric oxideischemia-reperfusionleft ventriclecoronary artery diseaseCardiac surgeryCardioprotectionCADarea at riskcyclic guanosine monophosphateNitrogen dioxideNO2Nitric oxide
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Authors
Yasuko Nagasaka, Bernadette O. Fernandez, Andrea U. Steinbicker, Ester Spagnolli, Rajeev Malhotra, Donald B. Bloch, Kenneth D. Bloch, Warren M. Zapol, Martin Feelisch,