| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 10871745 | FEBS Letters | 2011 | 6 Pages |
Abstract
⺠A variant VirB8 of B. suis with a point mutation G59W in the transmembrane domain has been studied. ⺠This mutation increases the stability of homodimers of this protein and heterodimers with VirB10. ⺠This variant is unable to restore the virulence of a virB8 deleted B. suis mutant. ⺠Similar results are found with the transmembrane variant A55W of A. tumefaciens VirB8. ⺠Interactions of VirB8 with VirB10 are likely as important as homodimerization of VirB8 for virulence.
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Authors
Leslie Andrieux, Gisèle Bourg, Audrey Pirone, David O'Callaghan, Gilles Patey,